Background Plaque erosion causes 30% of ST‐segment elevation myocardial infarctions, but the underlying cause is unknown. Inflammatory infiltrates are less abundant in erosion compared with rupture in autopsy studies. We hypothesized that erosion and rupture are associated with significant differences in intracoronary cytokines in vivo. Methods and Results Forty ST‐segment elevation

A 69-year-old male presented with medically refractory vertebrobasilar insufficiency and paroxysmal subjective dizziness for six months. Severe stenosis of a dominant left V2 vertebral artery segment was identified on digital subtraction angiography (DSA) with an irregular intraluminal filling defect immediately above the stenosis. Optical coherence tomography (OCT) demonstrated a normal lumen

Early evidence suggests that for patients with ACS caused by plaque erosion, noninterventional management with antithrombotic therapy without stenting may be an effective option without major adverse events. Plaque erosion is placed most in the proximal segment and near a bifurcation of the left anterior descending artery. Near a bifurcation is anatomically the most significant predictor of plaque erosion. Thrombus involved with plaque erosion occurs in fibrous or non-lipid plaques without TCFA and macrophages with larger lumen vessel.

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As the  Autopsy series identified nonruptured/eroded plaque and ruptured plaque as the principal pathological substrates underlying coronary thrombosis in STEMI. Project: Atherosclerotic plaque erosion leading to thrombus formation. The role of extracellular vesicles in ENDotheliaL transdiffErentiation, apoptoSiS and  Feb 22, 2021 Interview with Ik-Kyung Jang, MD, PhD, author of New Insights into Plaque Erosion as a Mechanism of Acute Coronary Syndromes. Apr 13, 2021 Plaque erosion deserves more attention as a distinct entity in patients with acute coronary syndromes—notably because it may not require  Dec 30, 2019 Plaque erosion is defined as an acute thrombus in direct contact with the intima in an area of denuded endothelium. Thus, plaque events ensue  Optical Coherence Tomography of Plaque Erosion and Thrombus in Severe Vertebral Artery Stenosis.

Download scientific diagram | Plaque rupture and plaque erosion in ACS. ( Modified from: Crea F, Liuzzo G: J Am Coll Cardiol. 2013;61(1):1-11, with permission 

Rafnsson,  Ut Gula Och Svarta Tänder Ont Dåliga Tänder Villkorar Erosion Karies Läkaren Plaque on teeth after smoking cigarettes and not observing toothbrushing  This Crest toothpaste formulation has the ADA Seal of Acceptance for controlling enamel erosion, gingivitis, and plaque, and for anti-sensitivity. Some toothpaste  The association of dental plaque with cancer mortality in Sweden. Larsen MJ, Nyvad B. Enamel erosion by some soft drinks and orange  ries prevalence, caries-related factors and plaque pH in adolescents with long- term asthma. Det finns också en ökad risk för dental erosion då barn och.

Isaksson H. On dental caries and dental erosion in Swedish young Birkhed D. Plaque pH and oral retention after consumption of starchy 

Transforming Growth Factor–β Signaling in T Cells Promotes Stabilization of Atherosclerotic Plaques Through an Interleukin-17–Dependent Pathway. Science  Papel (knottra) Plaque (palpabel med flat topp) Tidigare sjukdomar / Mediciner / Överkänslighet Nodulus (knuta, Storlek Erosion (under en sprucken blåsa) 5.

Our data suggest that there are differences in plaque formation or local thrombogenic potential in men and women with coronary artery disease. In the plaque-rupture group, 5 of 28 (18%) were women versus 11 of 22 (50%) with eroded plaques (P =.03). The mean percent luminal area stenosis was 78±12% in plaque rupture and 70±11% in superficial erosion (P <.03). Plaque calcification was present in 69% of ruptures versus 23% of erosions (P <.002). Plaque erosion is intimal injury which does not exhibit plaque rupture. Pathological studies have shown that plaque erosion often develops on intimal thickening or a fibroatheroma, with few lipid core and calcification.
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Thrombus involved with plaque erosion occurs in fibrous or non-lipid plaques without TCFA and macrophages with larger lumen vessel. Mechanisms of erosion have received much less attention than those that provoke plaque rupture.

Because the injured intima hides behind a thrombus, the plaque morphology is often overlooked by angioscopy. Objectives: The aim of this study was to characterize the morphological features of plaque erosion and calcified nodule in patients with acute coronary syndrome (ACS) by optical coherence tomography (OCT). Background: Plaque erosion and calcified nodule have not been systematically investigated in vivo. Plaque Erosion as a Mechanism of Acute Coronary Syndromes (ACS) JN Learning™ is the home for CME and MOC from the JAMA Network.
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A replica technique for studying the effect of fluoride solutions on enamel erosion Effect of nicotine-free and nicotine containing snus on plaque pH in vivo

Retrospective angiographic studies and the prospective PROSPECT (Providing Regional Observations to Study Predictors of Events in the Coronary Tree) study have shown that plaques with severe stenosis carry a higher per-plaque risk for producing clinical events than plaques that cause no or non severe stenosis. Plaque Erosionの機序: OPTICO-ACS. スポンサーリンク.

2019-10-12

An erosion, just like a plaque rupture, can cause the formation of a blood clot. However, because an erosion (in contrast to a rupture), is not an acute event, the clot often forms much more gradually. Although plaque rupture has been well-characterized by autopsy and clinical studies, current knowledge of plaque erosion remains poor.

2 More notably, neutrophils are often present. 3 But, autopsies cannot tell for sure whether the missing endothelium and neutrophil accumulation were the cause or result of thrombosis. This Plaque erosion is placed most in the proximal segment and near a bifurcation of the left anterior descending artery. Near a bifurcation is anatomically the most significant predictor of plaque erosion. Thrombus involved with plaque erosion occurs in fibrous or non-lipid plaques without TCFA and macrophages with larger lumen vessel. Differential accumulation of proteoglycans and hyaluronan in culprit lesions: insights into plaque erosion.